Hair

Better To Have Hair On Your Feet Than Your Head
A lack of hair on your feet may be a sign of circulatory problems, explains Andrew Manganaro, MD, Chief Medical Officer at Life Line Screening: When I was a resident surgeon, many years ago, one of my professors, himself a world renowned vascular surgeon, once said, "It is better to have hair on your feet than on your head." A receding hairline on your head may not be your favorite look, but lack of hair on your feet is much more dangerous. It is could be a sign that your feet are not getting adequate blood flow. The good news is that this can be checked with a simple screening that measures the blood pressure in your feet called an Ankle-Brachial Index. This screening can let you know if you are at risk for Peripheral Arterial Disease (PAD) or other circulatory problems. The Wall Street Journal's Melinda Beck also reports on the value of feet in evaluating circulatory health in her June 23rd article "What Your Body is Telling You." She writes, "Circulatory problems can manifest as numbness and tingling in the feet…When circulation is compromised, even a minor scratch or sore on the feet can become infected easily." I have always Said that HBOT can grow hair! Susan Rodriguez CHT #hbot Rapid Recovery Hyperbarics www.hbot4u.com

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Alycat780 has made a comment on Autism: child AFTER HBO at Rapid Recovery Hyperbarics!:
> Wow, what a gorgeous boy, and AMAZING progress!I love his huge grin, great eye contact and playful, adorable cuteness, he seems like such a happy and healthy cutie!!
> We, also experienced the ~WOW~ factor from HBOT, have done 40 hours so far...we are going back for another 10 hour booster soon, can't wait actually . Amazing how the healing power of oxygen combined with the body's innate drive to be in a constant ~heal itself~ mode, works wonders. I swear by HBOT, crazy, cool stuff. # autism www.hbot4u.com

Clinical pathological study of treatment of chronic hepatitis with hyperbaric oxygenation

 “Hepatitis C”

---

Liu W, Zhao W, Lu X, Zheng X, Luo C.Department of Gastroenterology, The Affiliated Nanjing Second Hospital of the Medical College of Nanjing Southeast University, Nanjing 210003, China
OBJECTIVE: To detect the feasibility and theoretic basis for treatment with hyperbaric oxygenation (HBOT) in chronic hepatitis and to compare the changes in hepatic function, immunity, pathologic morphology, ultra structure and HBV in hepatic tissues before and after treatment. METHODS: Sixty cases of chronic hepatitis were randomly selected and divided into two groups: the experiment (n = 30) and control groups (n = 30). Patients in the experimental group were treated with HBOT for 6 courses. Patients in the control group were treated for 60 days with the usual drugs used in the clinic. The function and bloodstream graph of liver were examined and liver biopsies were made before and after treatments. Routine paraffin sections were stained with HE and observed under the light microscope. Ultra thin slides from paraformaldehyde and glutaraldehyde fixed liver tissue were stained with lead citrate and observed with the transmission electric microscope. HBsAg and HBcAg in liver of the experimental group were detected with ABC immunohistochemistry method before and after treatment.

RESULTS: For the experimental group, ALT, SB, gamma-GT, AKP, IgG, and IgM in blood and the degeneration and necrosis of hepatocytes were remarkably decreased (P < 0.05 ), the mean contractive wave of bloodstream in liver and the bloodstream in right ramus of janitrix were remarkably increased (P < 0.05), and the swelling of mitochondria, increase of lysosomes, generation of Kupffer cells, infiltration of lymphocytes in portal area and capillary generation were all remarkably all eviated (P < 0.05) after treatment with HBOT. There were significant differences between the experimental and control groups after treatment with different methods (P < 0.05). For patients in the experimental group, the fibrosis and fat-storing cells in the liver were not reduced (P > 0.05), and the expression of HBsAg and HBcAg in liver was not weakened (P < 0.05) after treatment. 

CONCLUSIONS: Treatment with HBOT for chronic hepatitis was effective and recommendable, but it could not reverse liver fibrosis. However, it might be able to delay or prevent the liver from fibrosis, so it might be more effective at the early and middle stages of chronic hepatitis. HBOT could not inhibit the HB virus. So we consider that treatment with HBOT should be simultaneous with anti HBV therapy. Chin Med J (Engl). 2002 Aug;115(8):1153-7.

Reprinted with Permission: Rapid Recovery Hyperbarics www.hbot4u.com

The Magic Number 40

Forty sessions has come to be a benchmark in this therapy, not solely in the treatment of ASD, but rather in treating all conditions characterized by hypoxic tissue.  It is theorized that this is the number of treatments necessary to ensure angiogenesis has occurred.  In the past, physicians would notice “backsliding” or regression with most patients who had undergone less that thirty sessions.  On the other hand, patients having undergone forty sessions would not display symptoms of “backsliding”.  Today, with new knowledge regarding increased stem cell activity and sophisticated brain imaging equipment, it is easy to paint a picture as to what was occurring.  Recent research has demonstrated that after only ten sessions an eight fold increase in stem cell activity can be measured; in particular, CD34 stem cells which will produce new capillaries.  The formation of such capillaries can be seen between 15-20 sessions and the completion of such capillaries appears to occur between 30-40 sessions; hence the Magic Number 40.  Of course, this process is continually underway and new capillaries are just beginning at 20 sessions, 30 sessions, 40 sessions, etc.  One would assume that as long as hypoxic tissue remains in the body; stem cell activity would remain heightened, angiogenesis would continue, and tissue perfusion would continue to normalize.  In fact, in a study conducted with CP, stroke, and traumatic brain injury patients; it was demonstrated that throughout 70 sessions the rate of improvement in cerebral oxygenation increased during the last 35 treatments.  For this reason, it is not uncommon to see treatment protocols continue beyond the initial forty-session sequence; parents are often advised 60-80 initial sessions prior to pausing, ending, or lessening the frequency of treatment.

Frequency is Key and OXYGEN Pressure and Depth!~
There is much debate surrounding the optimal pressure in treating cerebral hypoperfusion related conditions such as Autism.  Case histories and research both report positive findings from pressures ranging 1.2 ATA to 2.0 ATA.  In addition, it becomes even more confusing as 100% oxygen is delivered Best results have been demonstrated through daily and twice daily treatment protocols.

Different Depths are designed for many disorders:

www.hbot4u.com 909-477-4545 Rapid Recovery Hyperbarics

HBOT Improves Peripheral Nerve Regeneration

Several studies have documented the effectiveness of hyperbaric oxygen in models of acute and delayed crush injury. Intermittent exposure to hyperbaric hyperoxia serves to interrupt the injury cycle of edema, ischemia and tissue necrosis ⁽¹⁾, as well as hemorrhagic hypotension ⁽²⁾, which in turn leads to former edema and ischemia. Tissue ischemia is countered by the ability of hyperbaric oxygen to elevate tissue oxygen tensions ⁽³⁾. Furthermore, edema is reduced, secondary to hyperoxia-induced arteriolar vasoconstriction ⁽⁴⁾, leading to improved tissue viability, thereby reducing necrosis ⁽¹⁾. Hyperbaric oxygen has also been studied in models of peripheral nerve injury ⁽⁵⁾. Researchers from the US Air Force School Aerospace Medicine and Louisiana State University recently sought to determine what, if any, morphologic changes are associated with hyperbaric oxygen treated peripheral nerve injury ⁽⁶⁾. Their model involved a crushed sciatic nerve in the rabbit.

Exposure to hyperbaric oxygen across the range of current clinical dose schedules was compared to untreated, and pressure (hyperbaric air) controls. A pathologist blinded as to group documented the extent of nerve regeneration via morphologic analysis of electron micrographs. All of the animals exposed to hyperbaric oxygen were reported to demonstrate advanced stages of a healed nerve, in contrast to both control groups. As this research was limited to a determination of regeneration of morphology, the exact effects of hyperbaric oxygen were not known. The authors speculate, however, that there may be several suggesting increased myelination, decreased edema, reduced internal collagen and improvements in neurofilamentous material density. They conclude that this study provides additional evidence of a link between tissue oxygen levels from hyperbaric oxygen treatment and the health of peripheral nerves.

... all animals exposed to hyperbaric oxygen "demonstrated characteristics expected of in the advanced stages of a healed nerve"

Of the estimated 20 million people in the United States with diabetes, 3 million struggle with DPN—and even patients with pre-diabetes and impaired glucose (blood sugar) tolerance may have symptoms. The older the person is, the longer he or she has had diabetes, and the less-controlled the disease, the greater chance of feeling pain because of damaged nerves.

Fifty percent of patients with long-standing diabetes have numbness, burning, electrical sensations, stabbing, or shooting pain in their feet or legs—and it’s usually worse at night. While the pain is uncomfortable, the lack of sensation can have even worse consequences. If a person’s shoes fit improperly, are too tight, have rough spots inside, or rub when the person walks, the blisters, abrasions or cuts may not be felt. Because circulation is not as good as it is for people without diabetes, these wounds can become infected and very difficult to heal.

Hyperbaric Oxygen Therapy is a very effective treatment for diabetic neuropathy. By driving oxygen deep into tissues, it reduces cell death and pain symptoms. Hyperbaric oxygen also stimulates the growth of new blood vessels, enabling the body to increase effective oxygen and nutrient delivery. #HBOT www.hbot4u.com

The Magic Number 40

Forty sessions has come to be a benchmark in this therapy, not solely in the treatment of ASD, but rather in treating all conditions characterized by hypoxic tissue.  It is theorized that this is the number of treatments necessary to ensure angiogenesis has occurred.  In the past, physicians would notice “backsliding” or regression with most patients who had undergone less that thirty sessions.  On the other hand, patients having undergone forty sessions would not display symptoms of “backsliding”.  Today, with new knowledge regarding increased stem cell activity and sophisticated brain imaging equipment, it is easy to paint a picture as to what was occurring.  Recent research has demonstrated that after only ten sessions an eight fold increase in stem cell activity can be measured; in particular, CD34 stem cells which will produce new capillaries.  The formation of such capillaries can be seen between 15-20 sessions and the completion of such capillaries appears to occur between 30-40 sessions; hence the Magic Number 40.  Of course, this process is continually underway and new capillaries are just beginning at 20 sessions, 30 sessions, 40 sessions, etc.  One would assume that as long as hypoxic tissue remains in the body; stem cell activity would remain heightened, angiogenesis would continue, and tissue perfusion would continue to normalize.  In fact, in a study conducted with CP, stroke, and traumatic brain injury patients; it was demonstrated that throughout 70 sessions the rate of improvement in cerebral oxygenation increased during the last 35 treatments.  For this reason, it is not uncommon to see treatment protocols continue beyond the initial forty-session sequence; parents are often advised 60-80 initial sessions prior to pausing, ending, or lessening the frequency of treatment.

Frequency is Key and OXYGEN Pressure and Depth!~
There is much debate surrounding the optimal pressure in treating cerebral hypoperfusion related conditions such as Autism.  Case histories and research both report positive findings from pressures ranging 1.2 ATA to 2.0 ATA.  In addition, it becomes even more confusing as 100% oxygen is delivered Best results have been demonstrated through daily and twice daily treatment protocols.

Different Depths are designed for many disorders:

www.hbot4u.com 909-477-4545 Rapid Recovery Hyperbarics

Contact us for a FREE informaitonal package!

Interested in Hyperbaric Oxygen Therapy? Call our office at 909.477.4545 or submit a contact form on our website www.hbot4u.com and we will send a FREE informational package to your doorstep about how Hyperbaric Oxygen Therapy at Rapid Recovery Hyperbarics can help you or your loved one's specific condition!

We look forward to talking with you!
-RRH

hbot4u.com
909.477.4545

Chronic Fatigue Syndrome Treatment

Sufferers of chronic fatigue syndrome have tried months and even years of various treatments, including taking pain medications and antidepressants, to obtain relief from this disabling condition. Hyperbaric oxygen therapy has shown promise in this frustrating medical condition and allowed many to return to their normal lifestyle.

Chronic Lyme Disease Treatment

The areas in and around Santa Monica and Malibu are well known to harbor the deer ticks capable of transmitting Lyme disease. In the chronic form of this disease—which has a multitude of symptoms including memory problems, fatigue and diffuse body pain—conventional medical treatment, including prolonged antibiotic therapy, has been disappointing in most patients. Hyperbaric oxygen therapy has been advocated by medical experts in the field of chronic Lyme disease, and many patients have found Hyperbaric Oxygen to be very beneficial. #lyme #hbot www.hbot4u.com #lyme #HBOT #rapidrecoveryHyperbarics

contact information

If you would like a full packet of information regarding our services, please send an email to info@hbot4u.com or call our office at 909.477.4545 We look forward to speaking to you..
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Rapid Recovery Hyperbarics
www.hbot4u.com

HBOT Decreases Inflammation in RSD

Hyperbaric oxygen treatment decreases inflammation and mechanical hypersensitivity in an animal model of inflammatory pain RSD

Wilson HD, Wilson JR, Fuchs PN
Brain Res
vol. 1098, 126 - 128, 2006

Abstract

Background: 

Hyperbaric treatment was initially developed to combat the adverse effects of deep sea diving. In recent years, hyperbaric oxygen treatment has been used to treat a broad spectrum of ailments, including delayed-onset muscle soreness, fibromyalgia, and complex regional pain syndrome (CRPS). However, limited data are available on the effect of hyperbaric oxygen treatment on inflammatory pain. The effect of hyperbaric oxygen treatment on carrageenan-induced inflammation and pain in rats was investigated. It was hypothesized that treatment with hyperbaric oxygen would decrease paw edema and hyperalgesia in an acute inflammatory pain model as compared with that of a sham-treated control group.

Methods: 

The experiment was conducted on 44 male Sprague-Dawley rats each between 300 and 350 g. The inflammatory pain condition was induced by subcutaneous injection of 1% carrageenan suspended in saline in the left hind paw. Paw volume was assessed via water displacement with a plethysmometer, and percentage differences were calculated on the basis of posttreatment measures compared with pretreatment measures. Hyperalgesia was assessed using the up/down method of mechanical paw withdrawal thresholds. Hyperbaric oxygen treatment involved exposing animals to 100% oxygen at a pressure of 2.4 atmospheres absolute (ATA) for 90 minutes in a hyperbaric chamber. A control group was placed in the hyperbaric chamber but did not receive treatment.

Results: 

A 1-way analysis of variance revealed no significant preinjec-tion group differences. An overall mixed-design analysis with 2 group levels (treatment, sham) and 7 levels of time revealed a significant main effect for group, a main effect for time, and a group by time interaction. In the hyperbaric oxygen treatment group, paw edema remained at pretreatment levels immediately after treatment and continued to decrease slightly until 2 hours posttreatment, at which time it began to decrease. However, antinociceptive effects were apparent immediately after treatment and continued to increase up to 5 hours after treatment, suggesting that distinct mechanisms might be involved in the anti-inflammatory and antinociceptive properties of Hyperbaric Oxygen Treatment.

Conclusions: 

Hyperbaric oxygen treatment significantly reduced inflammation and pain after carrageenan injection in this rat model of inflammatory pain. Hyperbaric oxygen may be used in patients for whom nonsteroidal anti-inflammatory drugs are contraindicated or for those with persistent inflammation.