Monday, May 30, 2011

Regarding Ear Equalization in HBOT Chamber

Regarding ear equalization with in a Hyperbaric Oxygen Chamber

Eustachian tube

Eustachian Tube
Ear-anatomy-text-small-en.svg
Anatomy of the human ear.
The middle ear
Latin tuba auditiva; tuba auditoria; tuba auditivea
Gray's subject #230 1042
Precursor first branchial pouch
MeSH Eustachian+tube

Babies’ Brains Process Words in an Adult Way

Babies’ Brains Process Words in an Adult Way
Babies process words using the same brain structures as adults, and in the same amount of time, according to a study by the University of California, San Diego. They are also able to understand words as more than simple sounds and comprehend the meanings of many of the words they hear.

“Babies are using the same brain mechanisms as adults to access the meaning of words from what is thought to be a mental ‘database’ of meanings, a database which is continually being updated right into adulthood,” says first author Katherine E. Travis of the Department of Neurosciences and the Multimodal Imaging Laboratory.

For the study, scientists used MRI and MEG (scan that measures magnetic fields emitted by neurons in the brain) to non-invasively analyze brain activity in infants, ages 12 to 18 months.

It was previously assumed that babies process words with a completely different learning mechanism, and it was believed that learning begins primitively—later evolving into the more “adult way” of learning. It has been difficult for scientists to figure out which areas of the brain are most involved in language learning because there is a lack information on how this process works in the developing brain.

Although lesions in two brain areas — Broca’s and Wernicke’s — have long been associated with loss of language skills in adults, these areas seem to have little impact on language development in early childhood. Some scientists have addressed this anomaly by theorizing that the right hemisphere and inferior frontal regions are vital for childhood language development, and that the other language areas of adulthood become dominant only when language development has matured.

Others have hypothesized that the plasticity of an infant’s brain allows other regions to take over the work of language-learning if the left frontotemporal regions become damaged at a young age.

During the first part of the experiment, the babies listened to words accompanied with sounds that have similar acoustic properties, but no meaning, to see if the infants could determine the difference between the two.

In the second part, the researchers wanted to see if the babies were able to understand the meaning of these words. For example, babies were shown pictures of familiar objects and then heard words that were either the correct or incorrect names for these objects: a picture of a ball followed by the spoken word ball, versus a picture of a ball followed by the spoken word dog.

It was determined through brain images that the infants could detect the mismatch between a picture and a word, as shown by the degree of brain activity. An incorrectly matched word triggered a classic brain response located in the same left frontotemporal areas known to process word meaning in the adult brain. The tests were then given to adults to confirm if the same mismatched picture/word combinations shown to babies would create larger responses in left frontotemporal areas.

“Our study shows that the neural machinery used by adults to understand words is already functional when words are first being learned,” said Eric Halgren, Ph.D., professor of radiology in the School of Medicine.

“This basic process seems to embody the process whereby words are understood, as well as the context for learning new words.”

The scientists believe the results could affect future studies. For example, the development of brain imaging tests could diagnose whether a baby has normal word understanding even before he or she can talk. This could allow early prediction for language disabilities or autism.

Sunday, May 29, 2011

Crush Injuries and Hyperbaric Oxygen Treatment


Crush Injuries and Hyperbaric Oxygen Treatment
Crush injury
is simply any injury to your body that is a result of trauma. These injuries could range from minor contusions to severe injuries that could damage a limb. It could be several tissues such as the skin, the muscles and tendons to bone and joints. These can result in osteomyelitis, failing union of fractures, failed flaps and amputations. The trauma can also result in skeleton muscle compartment syndrome (SMCS) which targets the muscles and the nerves. It is quite a challenge to manage the SMCS but it does not mean that it cannot be managed. This is where the crush injury hyperbaric becomes handy. It should be used as an adjunct to the management of the crush injuries or SMCS conditions.

We therefore need to consider the two ways by which the crush injury treatment that incorporates the hyperbaric oxygen works. First, it is important to note that during the initial stages of injury perfusion is likely to be inadequate. At such a time, immediate use of the hyperbaric oxygen makes more sense as it would supplement the supply of oxygen. The second way that really makes the use of hyperbaric oxygen useful is that it increases the tissue oxygen tension to the sufficient levels. Increased tissue oxygen in the plasma keeps the tissues alive without the oxygen that is borne from hemoglobin.

When the tension of tissue oxygen is increased, the effects of hypoxia that were initially felt when there was oxygen deficiency is countered. The other advantage that results from crush injury hyperbaric oxygen is the reduction of blood flow by up to 20 percent. The oxygen increases the induction of vasoconstriction and this is what causes the reduction. This in the end reduces edema. The filtration of the fluid that comes from the capillary to the extracellular space is also maintained.

The same treatment regime ensures that there is mitigation of reperfusion. This in turn interrupts the toxic oxygen radicals and the cell membrane. But you may ask how is that possible? Well, it actually perturbs lipid peroxiadation of the cell membrane and prevents the sequestration of neutrophils on the post capillary venules.

The other benefit of crush injury treatment hyperbaric is that it assists in provision of the oxygenated environment. Superoxide dismutase, catalase, peroxidase and glulathione which are useful for detoxification of reactive oxygen species actually depend in such environment to thrive.

When the crush injuries present in their severe forms, you should not think twice. You would be safe if you can quickly make use of the hyperbaric oxygen intervention that is quite effective. It is the only sensible and best way through which you would be able to counteract the pathophysiological events that occur when you have encountered a crush injury. The intervention is the sure way through which you will reduce loss of the muscle functions, the metabolites that come with muscle injury, edema and even muscle necrosis. Of course, this treatment is to be used together with the usual surgical and medical interventions that are in place.

Saturday, May 28, 2011

Autism changes molecular structure of the brain, UCLA study finds

Discovery points to a common cause for multifaceted disease By Elaine Schmidt May 25, 2011

For decades, autism researchers have faced a baffling riddle: how to unravel a disorder that leaves no known physical trace as it develops in the brain.

Now a UCLA study is the first to reveal how the disorder makes its mark at the molecular level, resulting in an autistic brain that differs dramatically in structure from a healthy one. Published May 25 in the advance online edition of Nature, the findings provide new insight into how genes and proteins go awry in autism to alter the mind.

The discovery also identifies a new line of attack for researchers, who currently face a vast array of potential fronts for tackling the neurological disease and identifying its diverse causes.

"If you randomly pick 20 people with autism, the cause of each person's disease will be unique," said principal investigator Dr. Daniel Geschwind, the Gordon and Virginia MacDonald Distinguished Chair in Human Genetics and a professor of neurology and psychiatry at the David Geffen School of Medicine at UCLA. "Yet when we examined how genes and proteins interact in autistic people's brains, we saw well-defined shared patterns. This common thread could hold the key to pinpointing the disorder's origins."

The research team, led by Geschwind, included scientists from the University of Toronto and King's College London. They compared brain tissue samples obtained after death from 19 autism patients and 17 healthy volunteers. After profiling three brain areas previously linked to autism, the group zeroed in on the cerebral cortex, the most evolved part of the human brain.

The researchers focused on gene expression — how a gene's DNA sequence is copied into RNA, which directs the synthesis of cellular molecules called proteins. Each protein is assigned a specific task by the gene to perform in the cell.

By measuring gene-expression levels in the cerebral cortex, the team uncovered consistent differences in how genes in autistic and healthy brains encode information.

"We were surprised to see similar gene expression patterns in most of the autistic brains we studied," said first author Irina Voineagu, a UCLA postdoctoral fellow in neurology. "From a molecular perspective, half of these brains shared a common genetic signature. Given autism's numerous causes, this was an unexpected and exciting finding."

The researchers' next step was to identify the common patterns. To do this, they looked at the cerebral cortex's frontal lobe, which plays a role in judgment, creativity, emotions and speech, and at its temporal lobes, which regulate hearing, language and the processing and interpreting of sounds.

When the scientists compared the frontal and temporal lobes in the healthy brains, they saw that more than 500 genes were expressed at different levels in the two regions.

In the autistic brains, these differences were virtually non-existent.

"In a healthy brain, hundreds of genes behave differently from region to region, and the frontal and temporal lobes are easy to tell apart," Geschwind said. "We didn't see this in the autistic brain. Instead, the frontal lobe closely resembles the temporal lobe. Most of the features that normally distinguish the two regions had disappeared."

Two other clear-cut patterns emerged when the scientists compared the autistic and healthy brains. First, the autistic brain showed a drop in the levels of genes responsible for neuron function and communication. Second, the autistic brain displayed a jump in the levels of genes involved in immune function and inflammatory response.

"Several of the genes that cropped up in these shared patterns were previously linked to autism," said Geschwind. "By demonstrating that this pathology is passed from the genes to the RNA to the cellular proteins, we provide evidence that the common molecular changes in neuron function and communication are a cause, not an effect, of the disease."

The next step will be for the research team to expand its search for the genetic and related causes of autism to other regions of the brain.

Autism is a complex brain disorder that strikes in early childhood. The disease disrupts a child's ability to communicate and develop social relationships and is often accompanied by acute behavioral challenges. In the United States, autism spectrum disorders are diagnosed in one in 110 children — and one in 70 boys. Diagnoses have expanded tenfold in the last decade.

The study was funded by the National Institute of Mental Health, the Canadian Institutes of Health Research, and Genome Canada. Tissue samples were provided by the Autism Tissue Project, the Harvard Brain Bank and the Medical Research Council's London Brain Bank for Neurodegenerative Disease.
Geschwind's and Voineagu's co-authors included Jennifer Lowe, Yuan Tian, Steve Horvath, Jonathan Mill, Rita Cantor and Benjamin Blencowe of UCLA; Xinchen Wang of the University of Toronto; and Patrick Johnston of King's College London.

Wednesday, May 25, 2011

Encephalitis and HBOT


Encephalitis and HBOT

Encephalitis simply means brain inflammation. The inflammatory reaction not only damages tissue, but also the microcirculation - small arteries capillaries and particularly small veins. The damage to the blood supply is the main determinant of recovery because of oxygen transport limitation.

The result is that brain tissue can remain in suspended animation "not dead but sleeping" - simply because of the increased tissue water - edema - chronically preventing adequate oxygenation. The best analogy is a comparison with an electrical device.

Imagine coupling a transistor radio - which requires a 9-volt battery to a 6-volt battery - it simply will not work - even if the six-volt battery is capable of delivering thousands of amperes - like a submarine battery.

There is nowhere on the surface of this planet that breathing air the oxygen level (voltage) in plasma can exceed about 100 (mm Hg).

In a hyperbaric chamber breathing pure oxygen, the level can be increased to over 2000 (mm Hg). This simple and completely scientific explanation for the need for hyperbaric conditions is UNKNOWN to the overwhelming majority of physicians - hence oxygen is not properly used.

Neurologists have recognized the need for oxygen. Three of the most eminent neurologists/neurosurgeons of the 80's stated in relation to the ischemic penumbra.

"Presumably the critical parameter for tissue function is oxygen availability rather than blood flow." Using hyperbaric conditions allows a decoupling of blood flow and oxygen transport.

Astrup J, Siesjo BK, Symon L. "Thresholds in cerebral ischemia -the ischemic penumbra." Stroke 1981;12:723-725.

Therefore, if we are to discover if there is recoverable brain tissue we need to reoxygenate. Dr Richard Neubauer has used SPECT imaging to demonstrate this effect and the work is published e.g. Lancet March 3 1990.

Dr Philip James M.D.
Reprinted with Permission

Sunday, May 22, 2011

Autism & HBOT


Rapid Recovery Hyperbarics
www.hbot4u.com
909.477.4545

Autism and HBOT
There is an estimated 1 to 1.5 million Autistic individuals in the United States. With an annual growth rate of 11-16%, and a 172% increase from the 1990’s, Autism is the fastest growing developmental disability in this country. The condition effects communication and social interactions and is the effect of neurological dysfunction. Autism is a spectrum disorder, with varying degrees of impact and is the most common of the Pervasive Developmental Disorders (PDD). Other disorders in this spectrum range include; Asperger's Syndrome, Rett's Disorder, Childhood Disintegrative Disorder and Not Otherwise Specified Pervasive Development Disorder.Hyperbaric Oxygen Therapy used in AutismHyperbaric oxygen therapy (HBOT) has been utilized to treat Autism in many countries throughout the globe. The rationale behind using hyperbaric for Autism is that the treatments increase cerebral blood flow and thus oxygen is delivered to areas of the brain, which are thought to be oxygen deficient. Greater amounts of blood and oxygen begin to stimulate cerebral tissues and aid in recovery of idling neurons. HBOT also reduces excess fluids and swelling of brain tissues which aid in neurological function.HBOT is also used as a complementary therapy for the treatment of heavy metal detox for such materials as mercury. Hyperbaric assists in the metabolism of heavy metal removal. It can help a patient counteract the effects of heavy metal poisoning and helps body deal with toxins even as noxious as cyanide. It is often used in conjunction with chelation and other detox procedures to help support the body to deal with the impact commonly seen in the removal of heavy metals, mercury, toxins, and other contaminants.Worldwide, medical researchers are discovering promising results with the effects of hyperbaric therapy on persons with Autism.In recent studies, measuring the effects of hyperbaric oxygen therapy on Autistic children ages 1-11 yrs, researchers found a total effectiveness of hyperbaric in 93.6 % of study participants. Researchers theorized that HBOT was effective in combating autoimmunity and the viral aspects of these conditions when encephalitis was present.So the question is- how can hyperbaric help with Autism? Scientists have varying opinions on why the therapy is so successful as a treatment for the condition. Some theories suggest that the brain in some persons with Autism, as well as Cerebral Palsy and other neurological disorders, is lacking oxygen and in a dormant, or sleeping state due to lack of blood flow. Hyperbaric forces oxygen into tissues throughout the body including brain tissues and fluid, resulting in a re-awakening of dormant areas of the brain. Other theories state that excessive swelling in the brain results in a lack of oxygen causing cerebral impairment that can cause behavioral problems, confusion, etc. Oxygen is forced into these damaged areas, through the pressurization of hyperbaric, which alleviates brain swelling by constricting blood vessels. In addition, hyperbaric is thought to provide an optimal environment to assist in the regeneration of brain tissue.

Rapid Recovery @ the 2011 I.E. Holistic Expo!




















Come out and visit the Rapid Recovery Hyperbarics booth at the 2011 Inland Empire Holistic Expo!!!

July 16th 2011

10am-6pm

Rancho Cucamonga Central Park Community Center

11200 Basline Road

Rancho Cucamonga CA 91701

Cost: $10

We can't wait to meet you!

Thursday, May 19, 2011

Brain Injury & Recovery with HBOT

Brain Injury and Recovery with Hyperbaric Oxygen

Hyperbaric Oxygen simply means oxygen given at increased barometric pressure.
The Problem:
The complex and almost continuous electrical activity of the brain is so discreet that we are unaware that it is the mechanism behind communication and thus intellectual and motor function. Brain injury can lead to a blockage of the electrical pathways.
Depending on the location of the injury, the brain's attempts to re-route through blocked pathways may cause frustrated discharges of activity known as seizures.


What Causes the Blockage?
SPECT scans (computerized brain mapping) show that not only does brain injury produce cell death, but it also reduces essential blood flow to a wider area of brain tissue surrounding the dead cells where signal re-routing might be expected to take place.
How Does This Happen?
After brain injury many blood capillaries around the area of cell death become torn open. The liquid part of the blood (the plasma) then leaks out, causing a swelling that may be very extensive. This reduces cerebral blood flow in the affected areas.
Reduction in blood flow means a reduction of essential nutrition (most vital oxygen), and a build up of waste products from local biochemical reactions (e.g. lactate and calcium), which shuts down normal cell function and further blocks pathways.
Why Doesn't Capillary Healing Happen?
If the capillaries are to heal, they desperately need oxygen. Unfortunately, the tiny tubules leading to the torn capillaries become constricted because of the damage.
This means that the red blood cells needed to bring the healing oxygen are too big to get through and simply get stuck in the "pipes." Thus, the plasma that is normally very low in oxygen continues to pour out, maintaining the swelling with all its related problems which, if left unattended, would last for a lifetime
Printed with Permission

HBOT & Crohn's Disease


Hyperbaric oxygen for perianal Crohn's disease.

Lavy A, Weisz G, Adir Y, Ramon Y, Melamed Y, Eidelman S. Department of Gastroenterology, Rambam Medical Center, Haifa, Israel. Perianal involvement in Crohn's disease is common, distressing, and frequently refractory to treatment. Clinical features include painful induration and stenosis, discharging fistulas, and fissures. The pathogenesis of these lesions is unclear, but local ischemia and secondary anaerobic infection may play a role. Following three sporadic reports of successful treatment with hyperbaric oxygen (HBO), we undertook a trial of this method in 10 patients with refractory perianal disease. These patients' perianal Crohn's disease had not responded to treatment that included local medications, salicylates, corticosteroids, metronidazole, or 6-mercaptopurine were treated. Treatment was administered in a hyperbaric chamber at a pressure of 2.5 atm absolute. Each session lasted 90 min, and each course consisted of 20 daily sessions. Complete healing occurred in 5 patients after one to two courses. In an additional 2, after three courses, 1 patient improved but did not heal, and 2 did not improve. No adverse effects were noted by any of the 10 patients. Follow-up of 18 months did not reveal any recurrence.
These preliminary results confirm that HBOT therapy is a safe and efficient therapeutic option for perianal Crohn's disease.
PMID: 7806829 [PubMed - indexed for MEDLINE]

Hypoxia


Hypoxia

Hypoxia is a state of oxygen deficiency in the body, which is sufficient to cause an impairment of function. Hypoxia is caused by the reduction in partial pressure of oxygen, inadequate oxygen transport, or the inability of the tissues to use oxygen.

In brief, being drunk is kind of the same as being exposed to high altitude. In both cases, oxygen to your brain and muscles is reduced.

Hypoxic Hypoxia is a reduction in the amount of oxygen passing into the blood. It is caused by a reduction in oxygen pressure in the lungs, by a reduced gas exchange area, exposure to high altitude, or by lung disease. [This is the hypoxia that is a hazard to aviators.]

Pemic Hypoxia is defined as a reduction in the oxygen carrying capacity of the blood. It is caused by a reduction in the amount of hemoglobin in the blood or a reduced number of red blood cells. A reduction in the oxygen transport capacity of the blood occurs through blood donation, hemorrhage, or anemia. A reduction in the oxygen carrying capacity of the blood occurs through drugs, chemicals, or carbon monoxide. [This hypoxia usually experienced by smokers.]

Stagnant Hypoxia is an oxygen deficiency due to poor circulation of the blood or poor blood flow. Examples of this condition are high "G" forces, prolonged sitting in one position or hanging in a harness, cold temperatures, and positive pressure breathing. [This hypoxia usually experienced when sitting for hours in a boring class.]

Histotoxic Hypoxia is defined as the inability of the tissues to use oxygen. Examples are carbon monoxide and cyanide poisoning. Certain narcotics, chewing tobacco, and alcohol will prevent oxygen use by the tissues. [This hypoxia usually experienced after drinking too much.]

Dr James MD

Reprinted with Permission


Hypoxia
Hypoxia is a state of oxygen deficiency in the body, which is sufficient to cause an impairment of function. Hypoxia is caused by the reduction in partial pressure of oxygen, inadequate oxygen transport, or the inability of the tissues to use oxygen.
In brief, being drunk is kind of the same as being exposed to high altitude. In both cases, oxygen to your brain and muscles is reduced.
Hypoxic Hypoxia is a reduction in the amount of oxygen passing into the blood. It is caused by a reduction in oxygen pressure in the lungs, by a reduced gas exchange area, exposure to high altitude, or by lung disease. [This is the hypoxia that is a hazard to aviators.]
Pemic Hypoxia is defined as a reduction in the oxygen carrying capacity of the blood. It is caused by a reduction in the amount of hemoglobin in the blood or a reduced number of red blood cells. A reduction in the oxygen transport capacity of the blood occurs through blood donation, hemorrhage, or anemia. A reduction in the oxygen carrying capacity of the blood occurs through drugs, chemicals, or carbon monoxide. [This hypoxia usually experienced by smokers.]
Stagnant Hypoxia is an oxygen deficiency due to poor circulation of the blood or poor blood flow. Examples of this condition are high "G" forces, prolonged sitting in one position or hanging in a harness, cold temperatures, and positive pressure breathing. [This hypoxia usually experienced when sitting for hours in a boring class.]
Histotoxic Hypoxia is defined as the inability of the tissues to use oxygen. Examples are carbon monoxide and cyanide poisoning. Certain narcotics, chewing tobacco, and alcohol will prevent oxygen use by the tissues. [This hypoxia usually experienced after drinking too much.]
Dr James MD
Reprinted with Permission

Saturday, May 14, 2011

Vaccine & Brain Injury / Death Linked


Acute Encephalopathy Followed by Permanent Brain Injury or Death Associated With Further Attenuated Measles Vaccines: A Review of Claims Submitted to the National Vaccine Injury Compensation Program

Rapid Recovery Hyperbarics www.hbot4u.com

Received Jul 30, 1997; accepted Sep 23, 1997.

Robert E. Weibel*, Vito Caserta*, David E. Benor Dagger, and Geoffrey Evans*

From the * Division of Vaccine Injury Compensation, National Vaccine Injury Compensation Program, Health Resources and Services Administration, Public Health Service, Rockville, Maryland; and the DaggerOffice of the General Counsel, United States Department of Health and Human Services, Rockville, Maryland.

PEDIATRICS Vol. 101 No. 3 March 1998, pp. 383-387
http://pediatrics.aappublications.org/cgi/content/abstract/101/3/383


Objective. To determine if there is evidence for a causal relationship between acute encephalopathy followed by permanent brain injury or death associated with the administration of further attenuated measles vaccines (Attenuvax or Lirugen, Hoechst Marion Roussel, Kansas City, MO), mumps vaccine (Mumpsvax, Merck and Co, Inc, West Point, PA), or rubella vaccines (Meruvax or Meruvax II, Merck and Co, Inc, West Point, PA), combined measles and rubella vaccine (M-R-Vax or M-R-Vax II, Merck and Co, Inc, West Point, PA), or combined measles, mumps, and rubella vaccine (M-M-R or M-M-R II, Merck and Co, Inc, West Point, PA), the lead author reviewed claims submitted to the National Vaccine Injury Compensation Program.

Methods. The medical records of children who met the inclusion criteria of receiving the first dose of these vaccines between 1970 and 1993 and who developed such an encephalopathy with no determined cause within 15 days were identified and analyzed.

Results. A total of 48 children, ages 10 to 49 months, met the inclusion criteria after receiving measles vaccine, alone or in combination. Eight children died, and the remainder had mental regression and retardation, chronic seizures, motor and sensory deficits, and movement disorders. The onset of neurologic signs or symptoms occurred with a nonrandom, statistically significant distribution of cases on days 8 and 9. No cases were identified after the administration of monovalent mumps or rubella vaccine.

Conclusions. This clustering suggests that a causal relationship between measles vaccine and encephalopathy may exist as a rare complication of measles immunization.
Key words: measles vaccine, encephalopathy, encephalitis.

Oxygen Deprivation May Contribute to Autism, Rapid Recovery Hyperbarics


Oxygen Deprivation May Contribute to Autism


MONDAY, Dec. 19 2009 (HealthDay News) -- New research with rats suggests that oxygen deprivation during birth could be a contributing cause of autism.

There's no easy way to test the oxygen-deprivation theory in humans, and the finding isn't likely to lead to better treatments in the near future. Still, the research gives scientists greater insight into how factors other than genetics may play a role in autism, said Fabrizio Strata, a neuroscience researcher at the University of California, San Francisco and co-author of the study.

Symptoms of autism, the most common condition in a group of developmental disorders known as autism spectrum disorders, can range from mild to severe. The disability usually strikes by age 3. It lasts a lifetime, and there is no cure, although some people with autism can learn to function well.

According to the U.S. Centers for Disease Control and Prevention, autism is characterized by three distinctive behaviors. Autistic children have difficulties with social interaction, display problems with verbal and nonverbal communication, and exhibit repetitive behaviors or narrow, obsessive interests. Scientists are not certain what causes autism, but it is likely that both genetics and environment play a role.

For reasons that aren't clear, autism seems to have become more common in recent years. One hotly debated theory suggests that vaccines are responsible, although some studies have failed to find a link.

Oxygen deprivation during birth is considered one possible cause because it can lead to brain damage.

By boosting the level of nitrogen in the air, Strata and colleagues deprived rat pups of normal levels of oxygen for as long as 10 to 12 minutes during birth. When the rats grew older, they displayed symptoms similar to those found in autistic children. It took longer for the rats to respond to some sounds, for example, and the brain regions that handle sound were disrupted.

Why would a baby be oxygen-deprived in the first place? According to Strata, a complicated labor can cut off a newborn's oxygen supply, as can a twisted umbilical cord.

Andy Shih, chief science officer with the National Alliance for Autism Research, said the oxygen-deprivation study presents an "interesting hypothesis," although the research hasn't been confirmed in humans.

It's possible that future research could lead to changes in obstetric practices to minimize the chance that babies will go without oxygen, Shih said. But "we're far away from that at this point."

The study findings appear in the Dec. 19-24 issue of the Proceedings of the National Academy of Sciences.


SOURCES: Fabrizio Strata, Ph.D., Keck Center for Integrative Neuroscience, University of California, San Francisco; Andy Shih, Ph.D., chief science officer, National Alliance for Autism Research, Princeton, N.J.; Dec. 19-23, 2005, Proceedings of the National Academy of Sciences

Tuesday, May 10, 2011

Exciting new things for RRH!!!


We are so excited for all the new things happening at Rapid Recovery Hyperbarics!
Our new location is opening soon, and a new website!
be sure to find us on facebook!- www.facebook.com/rapidrecoveryhyperbarics