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The use of HBOT after AGE on a 12 day old infant

Gas embolus and cardiac arrest during laparoscopic pyloromyotomy in an infant.

Department of Pediatric Anesthesiology, Children's Hospital of Wisconsin, Milwaukee, WI, USA, sutaylor@mcw.edu.

Abstract

PURPOSE: High volume tubing is used to deliver carbon dioxide during laparoscopic procedures. Failure to prime the tubing with carbon dioxide prior to abdominal insufflation may result in the delivery of nitrogen-containing air to the abdominal cavity. We report a case in which initial insufflation of laparoscopic gas resulted in immediate cardiovascular collapse requiring prolonged resuscitation. Persistent intracranial emboli following the arrest may have resulted from nitrogen contamination of the delivered gas. CLINICAL FEATURES: A 12-day-old female underwent laparoscopy for pyloric stenosis. During initial insufflation of the abdomen, the patient had an abrupt decrease in end-tidal carbon dioxide (CO(2ET)) associated with bradycardia and pulseless electrical activity. Three hours after successful resuscitation and open pyloromyotomy, computerized tomography documented intra-arterial gas within the cerebral and hepatic circulations that resolved following hyperbaric oxygen therapy. Magnetic resonance imaging five days later revealed watershed infarcts in the right frontal and parietal regions. Nitrogen, an insoluble gas not easily eliminated from the body, was likely the gas present within the patient's circulation several hours after the event. It was unlikely carbon dioxide, which is a highly soluble gas that binds to hemoglobin and is rapidly buffered by the carbonic anhydrase system and excreted by the lung. Room air contamination of high volume insufflation tubing allows nitrogen to enter body cavities during endoscopic procedures. CONCLUSION: Persistence of emboli following endoscopic procedures suggests that the entrained gas is insoluble. Room air contamination increases the potential for catastrophic events during laparoscopy and other endoscopic procedures.

PMID: 20431981 [PubMed - as supplied by publisher] #hbot #oxygen #rapidrecoveryHyperbarics www.hbot4u.com 9094774545 9439 Archibald Ave #104 Rancho Cucamonga, Ca 91730

16th year of service ! hbot4u.com

 This month we are celebrating our 16th year here at Rapid Recovery Hyperbarics!  Call 909 477 4545 visit our website www.hbot4u.com Follow Rapid Recovery Hyperbarics on facebook

 

  Anesthesia  >  Pain Management

Hyperbaric oxygen treatment decreases inflammation and mechanical hypersensitivity in an animal model of inflammatory pain RSD

Wilson HD, Wilson JR, Fuchs PN Brain Res vol. 1098, 126 - 128, 2006

Abstract

Background: 

Hyperbaric treatment was initially developed to combat the adverse effects of deep sea diving. In recent years, hyperbaric oxygen treatment has been used to treat a broad spectrum of ailments, including delayed-onset muscle soreness, fibromyalgia, and complex regional pain syndrome (CRPS). However, limited data are available on the effect of hyperbaric oxygen treatment on inflammatory pain. The effect of hyperbaric oxygen treatment on carrageenan-induced inflammation and pain in rats was investigated. It was hypothesized that treatment with hyperbaric oxygen would decrease paw edema and hyperalgesia in an acute inflammatory pain model as compared with that of a sham-treated control group.

Methods: 

The experiment was conducted on 44 male Sprague-Dawley rats each between 300 and 350 g. The inflammatory pain condition was induced by subcutaneous injection of 1% carrageenan suspended in saline in the left hind paw. Paw volume was assessed via water displacement with a plethysmometer, and percentage differences were calculated on the basis of posttreatment measures compared with pretreatment measures. Hyperalgesia was assessed using the up/down method of mechanical paw withdrawal thresholds. Hyperbaric oxygen treatment involved exposing animals to 100% oxygen at a pressure of 2.4 atmospheres absolute (ATA) for 90 minutes in a hyperbaric chamber. A control group was placed in the hyperbaric chamber but did not receive treatment.

Results: A 1-way analysis of variance revealed no significant preinjec-tion group differences. An overall mixed-design analysis with 2 group levels (treatment, sham) and 7 levels of time revealed a significant main effect for group, a main effect for time, and a group by time interaction. In the hyperbaric oxygen treatment group, paw edema remained at pretreatment levels immediately after treatment and continued to decrease slightly until 2 hours posttreatment, at which time it began to decrease. However, antinociceptive effects were apparent immediately after treatment and continued to increase up to 5 hours after treatment, suggesting that distinct mechanisms might be involved in the anti-inflammatory and antinociceptive properties of Hyperbaric Oxygen Treatment

.Conclusions: Hyperbaric oxygen treatment significantly reduced inflammation and pain after carrageenan injection in this rat model of inflammatory pain. Hyperbaric oxygen may be used in patients for whom nonsteroidal anti-inflammatory drugs are contraindicated or for those with persistent inflammation. www,hbot4u.com 909.477.4545

#hbot #RSD #CRPS #RAPIDRECOVERYHYPERBARICS #OXYGEN #HBOT

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Why can't I control my individual toes?

Bradley Voytek

Ph.D. neuroscience, UCSD Asst. Professor Cognit... (more) 

247 Votes by Paul King (Computational Neuroscientist, Redwood Center fo...), Aaron Kucyi (PhD Student in Neuroscience), Shan Kothari, and 244 more.

Before explaining this, I want you to try a quick experiment for me: hold your hand in front of you, fingers straight and pointing upward. Now, flex your index finger and justyour index finger. Did your middle finger flex, too? Maybe your ring finger even twitched a little? Try flexing just your ring finger. Unless you're a piano or string instrument player, it's unlikely that you were very successful at doing so.

The reason why that happens is closely related to why you can't control your individual toes. Stick with me.

This sexy beast is the motor homunculus:

He's built to reflect the relative area in the motor cortex that is devoted to controlling specific muscle groups. Notice how overrepresented the hands, lips, and eyes are and how underrepresented the arms, legs, and feet are?

Here's the motor cortex in the brain:

Basically, the more motor cortical area devoted to a region, the greater and finer the voluntary control over those muscles groups that we have.

Originally this map was created by Canadian neurosurgeon Wilder Penfield in 1937. Penfield pioneered brain surgery on awake patients. He  would use a small electrical stimulator to map out different parts of the brain, which is still done by neurosurgeons to this day. The logic was simple: stimulate a part of the motor cortex and watch which parts of the body twitched. This gives a mapping between brain and body, and what he found was a clear topography in the motor cortex.

Journalist and science writer Mo Costandi wrote an amazing history of Penfield here that's well worth reading, by the way:
http://scienceblogs.com/neurophi...

As I said in my answer to:

Are all the wrinkles on a brain's cortex the same across people?

...neurosurgeons will perform electrical stimulation mapping of awake people if they have to remove any brain tissue near what they call "eloquent cortex"... [because] [t]he only way even an experienced surgeon can be sure that specific brain area in a specific person is motor, or speech, or sensory, is via this mapping technique... This is because, although gross neuroanatomical features are generally conserved across people, there can be a huge range of variation.

Penfield used a highly invasive means to map out the motor homunculus. But it turns out we have some pretty cool modern technology with which we study the motor cortex non-invasively: Transcranial Magnetic Stimulation (TMS).

TMS induces an electrical current using a rapidly changing magnetic field

MS

Multiple Sclerosis and HBOT

The number of Americans suffering from the debilitating effects of Multiple Sclerosis is estimated to be a quarter of a million and growing. MS is a central nervous system disease that is thought to be an autoimmune condition generated by a virus, although definite causes are unknown at this time. The condition is characterized as a relapsing and remitting disease that is aggravated by stress and an abnormal immune system, which results in inflammation, damage and hardening of myelin and nerve fibers that typically occurs in the spinal cord, brain stem and optic nerves.

Multiple Sclerosis symptomology includes weakness, severe pain, loss of vision, and extreme pain, which are caused by slowed nerve impulses as a result of damage to the nerves or myelin, which is known as demyelination. Persons with MS also suffer from balance problems, bladder and bowel dysfunction, difficulty walking, vision loss and pain in eyes, depression, neurological dysfunction, spasticity of muscles, and debilitating fatigue.

Hyperbaric Oxygen Therapy

Extensive international medical research, over the past twenty years, has demonstrated that hyperbaric oxygen therapy will be considered an integral role in the treatment of Multiple Sclerosis. In many parts of the world, hyperbaric is considered a key component of an effective MS treatment program. In some countries in Europe, hyperbaric oxygen therapy is covered by insurance and is the main treatment for MS.

Some theories suggest that Multiple Sclerosis could be attributed to a lack of oxygen in the body, resulting in the lesions that appear on the spinal cord of patients undergoing SPECT scans. Regardless of what the causes of MS are, there is irrefutable evidence supporting the treatment of MS patients with hyperbaric oxygen. More and more medical researchers have come to regard hyperbaric oxygen therapy is a potent therapy for the successful treatment of multiple sclerosis without the side effects associated with many pharmaceuticals. Hyperbaric has been shown to reduce pain significantly, modulate the immune system, increase energy, alleviate sleep dysfunction, and reduce cognitive impairment. In most cases, patients are also able to discontinue use of many of their pharmaceuticals. Hyperbaric oxygen therapy raises oxygen levels in the body by up to 1000% through the increased pressure of the HBO chamber. Viruses are unable to survive in the presence of abundant oxygen, therefore a significant “die-off” of viruses and bacteria is experienced when undergoing hyperbaric treatments. Hyperbaric also acts as an immune modulator and allows organ and gland functionality to normalize, reducing many debilitating MS symptoms. Because of the pressurized environment inside the chamber, hyperbaric acts as a detoxifier as well. By forcing oxygen into the tissues through this pressure, toxins, chemicals and other impurities are forced out.

A recent study sited by the New England Journal of Medicine demonstrated compelling evidence of improvements in MS symptomology. Forty chronic Multiple Sclerosis patients, in the advanced stages of the disease, were treated with hyperbaric oxygen therapy at 2 ATA for 90 minute sessions. Seventy percent of those treated received relief from the effects of MS after the 20 study treatments. Patients in the study who received the greatest benefit from the hyperbaric were those who had a less severe form of MS, however, because the study consisted of only 20 treatments, the results on more severe MS were not as evident.

Some medical professionals state that the most effective protocol for hyperbaric oxygen on persons with MS is that of lower pressures at 1.5 ATA or below for longer treatment times. #MS #HBOT www.hbot4u.com 909.477.4545

Hair

Better To Have Hair On Your Feet Than Your Head
A lack of hair on your feet may be a sign of circulatory problems, explains Andrew Manganaro, MD, Chief Medical Officer at Life Line Screening: When I was a resident surgeon, many years ago, one of my professors, himself a world renowned vascular surgeon, once said, "It is better to have hair on your feet than on your head." A receding hairline on your head may not be your favorite look, but lack of hair on your feet is much more dangerous. It is could be a sign that your feet are not getting adequate blood flow. The good news is that this can be checked with a simple screening that measures the blood pressure in your feet called an Ankle-Brachial Index. This screening can let you know if you are at risk for Peripheral Arterial Disease (PAD) or other circulatory problems. The Wall Street Journal's Melinda Beck also reports on the value of feet in evaluating circulatory health in her June 23rd article "What Your Body is Telling You." She writes, "Circulatory problems can manifest as numbness and tingling in the feet…When circulation is compromised, even a minor scratch or sore on the feet can become infected easily." I have always Said that HBOT can grow hair! Susan Rodriguez CHT #hbot Rapid Recovery Hyperbarics www.hbot4u.com

CHeck out our Youtube video

Alycat780 has made a comment on Autism: child AFTER HBO at Rapid Recovery Hyperbarics!:
> Wow, what a gorgeous boy, and AMAZING progress!I love his huge grin, great eye contact and playful, adorable cuteness, he seems like such a happy and healthy cutie!!
> We, also experienced the ~WOW~ factor from HBOT, have done 40 hours so far...we are going back for another 10 hour booster soon, can't wait actually . Amazing how the healing power of oxygen combined with the body's innate drive to be in a constant ~heal itself~ mode, works wonders. I swear by HBOT, crazy, cool stuff. # autism www.hbot4u.com

Clinical pathological study of treatment of chronic hepatitis with hyperbaric oxygenation

 “Hepatitis C”

---

Liu W, Zhao W, Lu X, Zheng X, Luo C.Department of Gastroenterology, The Affiliated Nanjing Second Hospital of the Medical College of Nanjing Southeast University, Nanjing 210003, China
OBJECTIVE: To detect the feasibility and theoretic basis for treatment with hyperbaric oxygenation (HBOT) in chronic hepatitis and to compare the changes in hepatic function, immunity, pathologic morphology, ultra structure and HBV in hepatic tissues before and after treatment. METHODS: Sixty cases of chronic hepatitis were randomly selected and divided into two groups: the experiment (n = 30) and control groups (n = 30). Patients in the experimental group were treated with HBOT for 6 courses. Patients in the control group were treated for 60 days with the usual drugs used in the clinic. The function and bloodstream graph of liver were examined and liver biopsies were made before and after treatments. Routine paraffin sections were stained with HE and observed under the light microscope. Ultra thin slides from paraformaldehyde and glutaraldehyde fixed liver tissue were stained with lead citrate and observed with the transmission electric microscope. HBsAg and HBcAg in liver of the experimental group were detected with ABC immunohistochemistry method before and after treatment.

RESULTS: For the experimental group, ALT, SB, gamma-GT, AKP, IgG, and IgM in blood and the degeneration and necrosis of hepatocytes were remarkably decreased (P < 0.05 ), the mean contractive wave of bloodstream in liver and the bloodstream in right ramus of janitrix were remarkably increased (P < 0.05), and the swelling of mitochondria, increase of lysosomes, generation of Kupffer cells, infiltration of lymphocytes in portal area and capillary generation were all remarkably all eviated (P < 0.05) after treatment with HBOT. There were significant differences between the experimental and control groups after treatment with different methods (P < 0.05). For patients in the experimental group, the fibrosis and fat-storing cells in the liver were not reduced (P > 0.05), and the expression of HBsAg and HBcAg in liver was not weakened (P < 0.05) after treatment. 

CONCLUSIONS: Treatment with HBOT for chronic hepatitis was effective and recommendable, but it could not reverse liver fibrosis. However, it might be able to delay or prevent the liver from fibrosis, so it might be more effective at the early and middle stages of chronic hepatitis. HBOT could not inhibit the HB virus. So we consider that treatment with HBOT should be simultaneous with anti HBV therapy. Chin Med J (Engl). 2002 Aug;115(8):1153-7.

Reprinted with Permission: Rapid Recovery Hyperbarics www.hbot4u.com

The Magic Number 40

Forty sessions has come to be a benchmark in this therapy, not solely in the treatment of ASD, but rather in treating all conditions characterized by hypoxic tissue.  It is theorized that this is the number of treatments necessary to ensure angiogenesis has occurred.  In the past, physicians would notice “backsliding” or regression with most patients who had undergone less that thirty sessions.  On the other hand, patients having undergone forty sessions would not display symptoms of “backsliding”.  Today, with new knowledge regarding increased stem cell activity and sophisticated brain imaging equipment, it is easy to paint a picture as to what was occurring.  Recent research has demonstrated that after only ten sessions an eight fold increase in stem cell activity can be measured; in particular, CD34 stem cells which will produce new capillaries.  The formation of such capillaries can be seen between 15-20 sessions and the completion of such capillaries appears to occur between 30-40 sessions; hence the Magic Number 40.  Of course, this process is continually underway and new capillaries are just beginning at 20 sessions, 30 sessions, 40 sessions, etc.  One would assume that as long as hypoxic tissue remains in the body; stem cell activity would remain heightened, angiogenesis would continue, and tissue perfusion would continue to normalize.  In fact, in a study conducted with CP, stroke, and traumatic brain injury patients; it was demonstrated that throughout 70 sessions the rate of improvement in cerebral oxygenation increased during the last 35 treatments.  For this reason, it is not uncommon to see treatment protocols continue beyond the initial forty-session sequence; parents are often advised 60-80 initial sessions prior to pausing, ending, or lessening the frequency of treatment.

Frequency is Key and OXYGEN Pressure and Depth!~
There is much debate surrounding the optimal pressure in treating cerebral hypoperfusion related conditions such as Autism.  Case histories and research both report positive findings from pressures ranging 1.2 ATA to 2.0 ATA.  In addition, it becomes even more confusing as 100% oxygen is delivered Best results have been demonstrated through daily and twice daily treatment protocols.

Different Depths are designed for many disorders:

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HBOT Improves Peripheral Nerve Regeneration

Several studies have documented the effectiveness of hyperbaric oxygen in models of acute and delayed crush injury. Intermittent exposure to hyperbaric hyperoxia serves to interrupt the injury cycle of edema, ischemia and tissue necrosis ⁽¹⁾, as well as hemorrhagic hypotension ⁽²⁾, which in turn leads to former edema and ischemia. Tissue ischemia is countered by the ability of hyperbaric oxygen to elevate tissue oxygen tensions ⁽³⁾. Furthermore, edema is reduced, secondary to hyperoxia-induced arteriolar vasoconstriction ⁽⁴⁾, leading to improved tissue viability, thereby reducing necrosis ⁽¹⁾. Hyperbaric oxygen has also been studied in models of peripheral nerve injury ⁽⁵⁾. Researchers from the US Air Force School Aerospace Medicine and Louisiana State University recently sought to determine what, if any, morphologic changes are associated with hyperbaric oxygen treated peripheral nerve injury ⁽⁶⁾. Their model involved a crushed sciatic nerve in the rabbit.

Exposure to hyperbaric oxygen across the range of current clinical dose schedules was compared to untreated, and pressure (hyperbaric air) controls. A pathologist blinded as to group documented the extent of nerve regeneration via morphologic analysis of electron micrographs. All of the animals exposed to hyperbaric oxygen were reported to demonstrate advanced stages of a healed nerve, in contrast to both control groups. As this research was limited to a determination of regeneration of morphology, the exact effects of hyperbaric oxygen were not known. The authors speculate, however, that there may be several suggesting increased myelination, decreased edema, reduced internal collagen and improvements in neurofilamentous material density. They conclude that this study provides additional evidence of a link between tissue oxygen levels from hyperbaric oxygen treatment and the health of peripheral nerves.

... all animals exposed to hyperbaric oxygen "demonstrated characteristics expected of in the advanced stages of a healed nerve"

Of the estimated 20 million people in the United States with diabetes, 3 million struggle with DPN—and even patients with pre-diabetes and impaired glucose (blood sugar) tolerance may have symptoms. The older the person is, the longer he or she has had diabetes, and the less-controlled the disease, the greater chance of feeling pain because of damaged nerves.

Fifty percent of patients with long-standing diabetes have numbness, burning, electrical sensations, stabbing, or shooting pain in their feet or legs—and it’s usually worse at night. While the pain is uncomfortable, the lack of sensation can have even worse consequences. If a person’s shoes fit improperly, are too tight, have rough spots inside, or rub when the person walks, the blisters, abrasions or cuts may not be felt. Because circulation is not as good as it is for people without diabetes, these wounds can become infected and very difficult to heal.

Hyperbaric Oxygen Therapy is a very effective treatment for diabetic neuropathy. By driving oxygen deep into tissues, it reduces cell death and pain symptoms. Hyperbaric oxygen also stimulates the growth of new blood vessels, enabling the body to increase effective oxygen and nutrient delivery. #HBOT www.hbot4u.com